WebChlamydia is a sexually transmitted disease and is caused by Chlamydia trachomatis, a gram-negative bacterium but not a spirochete. Chlamydiae are obligate, intracellular, nonmotile human pathogen that causes inclusion conjunctivitis, lymphogranuloma …View the … WebChlamydia trachomatis (CT) is an obligate intracellular organism that is Gram-negative, ovoid in shape, and nonmobile. Most genital isolates belong to immunotypes D through …
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WebMar 14, 2024 · Chlamydia trachomatis is an obligate intracellular bacterium that is responsible for the most prevalent bacterial sexually transmitted infections. Changes in DNA topology in this pathogen have been linked to its pathogenicity-associated developmental cycle. Here, evidence is provided that the balanced activity of DNA topoisomerases … WebPathogens and Disease are proud to present our latest Thematic Issue on Obligate Intracellular Bacterial Pathogens. In this thematic issue, we compiled a collec. ... In this article, the authors review the function of Chlamydia trachomatis secreted effector proteins and Incs, with special emphasis on how the advancement of chlamydial genetics ... phil sexton
Chlamydia trachomatis Infections - Ohio
WebIntroductionChlamydia trachomatis, a gram-negative obligate intracellular bacterium, commonly causes sexually transmitted infections (STIs). Little is known about C. trachomatis transmission within the host, which is important for understanding disease epidemiology and progression.MethodsWe used RNA-bait enrichment and whole … WebThe obligate intracellular grown bacterium Chlamydia trachomatis causes diseases like trachoma, urogenital infection and lymphogranuloma venereum with severe morbidity. … WebChlamydia trachomatis is an obligate intracellular parasite that elaborates antigens on its surface. These antigens are divided into genus-, species-, subspecies-, and serovar-specific determinants. The genus, or group antigen(s), are lipopolysaccharides similar to those found in gram-negative bacteria and a glycolipid that is secreted by ... phil sewell facebook